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Eczema
is the most common skin condition seen in day-to-day practice.
It is a pattern of inflammatory response (dermatitis) of the skin
characterised by itching, redness, flaking of the skin and raised,
fluid filled lesions (papules and vesicles). The condition may
be induced by a wide variety of external and internal factors
acting alone or in combination.
Based on the
causes, eczema can be classified as exogenous eczema or
endogenous
eczema. A detailed history of a patient with eczema is very
important to identify the underlying cause/s, as long term remissions
can be achieved only by eliminating the underlying cause/s.
Sometimes, the tendency for eczema runs in the family and therefore
occurrence of eczema in family members does not imply that it
is contagious.
Exogenous causes:
Tendency for eczema can be
familial...
- Irritant dermatitis
(detergents, soaps, chemicals)
- Allergic dermatitis
(rubber, cement, dyes, plants)
- Photo-sensitive
dermatitis (dermatitis caused or precipitated by sunlight and
photosensitive agents)
- Polymorphic light
eruption (caused only by UV radiation present in the sunlight)
- Infective dermatitis
(dermatitis occurring in and around the infected wound)
Endogenous causes:
- Atopic (dermatitis
occurring in a person with personal or family history of asthma,
allergic rhinitis)
- Seborrhoeic
dermatitis (dermatitis seen in areas rich in sebaceous glands;
usually after puberty)
- Asteatotic
eczema (eczema associated with decrease in skin surface oils;
common in the elderly)
- Gravitational
eczema (eczema associated with varicose veins; seen around
the ankles)
- Hand eczema (most
common eczema; causes can be many)
- Forefoot eczema
(common eczema seen in childhood, involving predominantly the
forefoot)
- Pityriasis alba (dry,
white patches seen on the face and sometimes elsewhere;
occurs during
childhood)
- Metabolic eczema
(eczema due to nutritional deficiencies of EFA, zinc, niacin,
proteins)
- Drug eruptions (exfoliative
dermatitis)
Two or more types
of eczema may be present in the same patient simultaneously
or consecutively. Certain eczema are common in particular age
groups:
- Infants - atopic
- Childhood - atopic,
forefoot eczema, pityriasis alba
- Adolescence -
seborrhoic dermatitis, allergic and irritant dermatitis to cosmetics
- Middle age - Stasis
eczema, irritant and allergic dermatitis (occupational)
- Elderly - asteatotic
eczema
Based on the
clinical presentation, eczemas are termed as-
- Acute (when
there is oozing, with tiny fluid filled lesions and
swelling)
- Subacute
(scaly and red)
- Chronic
(thick and hyperpigmented skin)
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Asteatotic
eczema |
Asteatotic
eczema |
Diagnosis:
A detailed
history is important to determine the cause (exogenous/
endogenous) and precipitating factors. Eczemas have to be
differentiated from other common skin diseases like
- Tinea
(fungal infection/'ring worm')
- Psoriasis
- Lichen
planus etc.
Differentiation
from Tinea is very important as the treatment is entirely
different.
- Eczemas are
usually not sharply demarcated
- Eczemas do
not show clearance in the centre (unless partially treated)
- Eczemas are
usually bilateral and if unilateral, correspond to the site
of contact with exogenous agent (contact dermatitis)
- In acute
stage, eczema is characterised by oozing.
In case of
doubts, scraping should be taken from the scales to demonstrate
the fungal elements.
Patch testing
is required if clinical presentation (history and site of involvement)
is suggestive of an exogenous cause. It is also indicated if eczema
of endogenous origin does not respond to treatment or is persistent
in spite of treatment.
Biopsy
is required in unusual cases and unusual presentations.
Treatment:
Treatment should
aim at removing or reversing the cause. Treatment should be
such that the patient should have a long term remission with
minimum or no side effects to the medications.
In the presence
of acute exacerbation, compresses (potassium permanganate)
should be given to reduce the oozing and oedema, following which
bland preparations should be applied. Topical steroids can be
used for quicker resolution. In the presence of infection (yellow
crusting) topical steroids with antibiotics (other than gentamicin
and neomycin) can be used. Oral antibiotics (targeting Streptococci
and Staphylococci) and antihistamines (to reduce
itching) may be required.
Subacute
eczema: Sometimes eczemas can present in this stage or acute
eczema after partial treatment may manifest in this form.
Treatment includes avoidance of rubbing, use of emolients/moisturizers
regularly during the day and topical steroids with or without
moisturizers in the evenings.
Eczema can be best controlled and relapse
prevented by avoiding the cause/s
Chronic
eczema: Long term rubbing and scratching of eczematous or
normal skin leads to chronic eczema. Treatment obviously
includes avoidance of rubbing or scratching, emolients/moisturizers
to protect and soothe the damaged skin during the day and
topical steroids with salicylic acid, urea, lactic acid in the
night. In addition, the area should be made inaccessible for
scratching by occlusion of the area with clothing.
Maintenance therapy
includes regular use of moisturizers, best suited for the type
and site of the skin.
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Points
to remember regarding eczema:
-
Do
not try self-medication or home remedies
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Do
not use potent topical steroids for long without
doctor's advice (not more than 2 weeks)
-
Avoid
the likely cause/precipitating factors
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Use
the treatment for the specified duration continuously
without interruption. Do not stop the treatment
after the itching disappears; continue treatment
until the skin comes back to its normal texture.
-
Topical
steroid is not the only form of treatment. It has
to be continued or replaced with emolients/moisturizers
simultaneously or later
-
Stress
can aggravate eczema
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